Which shock is associated with systemic infection leading to inflammation?

Septic shock is associated with systemic infection leading to inflammation, which is a critical aspect of its pathophysiology. In septic shock, the body's response to the infection results in widespread inflammation, causing a cascade of physiological changes. These changes include vasodilation, increased vascular permeability, and altered blood flow, which ultimately leads to inadequate tissue perfusion and oxygen delivery.

The presence of infection triggers the release of inflammatory mediators that exacerbate the response, potentially resulting in multi-organ dysfunction. This condition is characterized by low blood pressure and high levels of lactate in the bloodstream due to cellular hypoxia, further highlighting the systemic impact of the infection.

In contrast, other types of shock, such as hypovolemic or cardiogenic shock, are primarily related to inadequate volume or heart function, rather than an inflammatory response to infection. Aplastic shock, while less common, typically refers to bone marrow failure rather than an infectious cause. The distinct relationship between inflammation resulting from systemic infection and the hemodynamic instability in septic shock is what differentiates it from other shock states.